Stalled/collapsed replication forks from nucleotide shortage, DNA damage, R-loops. ATR-Chk1 response. Key cancer hallmark; targetable by ATR/Chk1 inhibitors.
Stalled/collapsed replication forks from nucleotide shortage, DNA damage, R-loops. ATR-Chk1 response. Key cancer hallmark; targetable by ATR/Chk1 inhibitors.